I have to say, I have always been a skeptic. One of the nurses I work with told me to give my son Vitamin D3. she was specific, saying that Vitamin D3 and Vitamin D2 have opposite effects on the aortic valve. Needless to say, I started doing research. I have now put our entire family on a dose of vitamin D3 of 400mg daily in hopes that the new information popping up all over the place might be correct.
You guys hear about this? Any thoughts?
Here are some of the articles I ran across... small excerpts and links...
Another case of aortic valve disease reduced with vitamin D
Dr. Davis writes:
“While Seth's last echocardiogram showed a severely leaky aortic valve, the most recent echo showed mild leakiness ("mild aortic insufficiency")--a dramatic reduction.
http://heartscanblog.blogspot.com/2008/12/another-case-of-aortic-valve-disease.html
More on aortic valve disease and vitamin D
Dr. William D. writes:
“I hope I'm not getting my hopes up prematurely, but I believe that I've seen it once again: Dramatic reversal of aortic valve disease.”
http://www.wellsphere.com/heart-health-article/more-on-aortic-valve-disease-and-vitamin-d/239730
The vitamin D receptor genotype predisposes to the development of calcific aortic valve stenosis
Part of the abstract states:
“There is a significant association of vitamin D receptor polymorphism with calcific aortic valve stenosis. The B allele of the vitamin D receptor is more common in patients with calcific aortic valve stenosis. It now needs to be evaluated whether other genes that control calcium homeostasis are involved in the pathogenesis of this disorder.”
http://heart.bmj.com/content/85/6/635.short
Vitamin D and the Supravalvar Aortic Stenosis Syndrome
An excerpt from this article is:
“Thirty-five control offspring and nine rabbits born to mothers on vitamin D-deficient diets showed no abnormalities of the aorta. The results suggest that an in utero derangement in vitamin D metabolism on the part of mother or fetus, or of both may be responsible for supravalvar aortic stenosis, especially when the latter is associated with infantile hypercalcemia.”
http://circ.ahajournals.org/cgi/content/abstract/34/1/77
Vitamin D2 induces aortic stenosis
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2439857/
You guys hear about this? Any thoughts?
Here are some of the articles I ran across... small excerpts and links...
Another case of aortic valve disease reduced with vitamin D
Dr. Davis writes:
“While Seth's last echocardiogram showed a severely leaky aortic valve, the most recent echo showed mild leakiness ("mild aortic insufficiency")--a dramatic reduction.
http://heartscanblog.blogspot.com/2008/12/another-case-of-aortic-valve-disease.html
More on aortic valve disease and vitamin D
Dr. William D. writes:
“I hope I'm not getting my hopes up prematurely, but I believe that I've seen it once again: Dramatic reversal of aortic valve disease.”
http://www.wellsphere.com/heart-health-article/more-on-aortic-valve-disease-and-vitamin-d/239730
The vitamin D receptor genotype predisposes to the development of calcific aortic valve stenosis
Part of the abstract states:
“There is a significant association of vitamin D receptor polymorphism with calcific aortic valve stenosis. The B allele of the vitamin D receptor is more common in patients with calcific aortic valve stenosis. It now needs to be evaluated whether other genes that control calcium homeostasis are involved in the pathogenesis of this disorder.”
http://heart.bmj.com/content/85/6/635.short
Vitamin D and the Supravalvar Aortic Stenosis Syndrome
An excerpt from this article is:
“Thirty-five control offspring and nine rabbits born to mothers on vitamin D-deficient diets showed no abnormalities of the aorta. The results suggest that an in utero derangement in vitamin D metabolism on the part of mother or fetus, or of both may be responsible for supravalvar aortic stenosis, especially when the latter is associated with infantile hypercalcemia.”
http://circ.ahajournals.org/cgi/content/abstract/34/1/77
Vitamin D2 induces aortic stenosis
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2439857/
