If you are coming here with a normal tricuspid valve and CALCIFIC DEGENERATIVE aortic valve stenosis, don’t let anyone here dump on you for having “done it to yourself”, as happened to me. High Lp(a) is determined by genetics and some people have shockingly high levels of they have more than one copy of a mutant allele that raises it (I have two and my Lp(a) is 13 times normal, triple the “extremely high risk” rate). If you have AVS from high Lp(a), it is likely that your AVS is only one facet of your health problems, as high Lp(a) drives coronary artery disease and other problems as well. Most people here have AVS because they were born with bicuspid valves, and don’t really understand about Lp(a). This is a place for THEM. There are other places for high Lp(a) patients, like the Lp(a) Foundation on Facebook. Find out about the new drugs coming out (Phase 3 trials started this year) that can lower Lp(a) by as much as 90%! Treatment that can slow the progression of your AVS is on the way!
Serial imaging showed it, and a lab experiment nailed it — at least in vitro. Elevations in two biomarkers further the progression of aortic valve calcification, and their inhibition might thwart it.